Eryngium Maritimum Callus Culture Filtrate

1. Definition Eryngium Maritimum Callus Culture Filtrate:

Eryngium Maritimum Callus Culture Filtrate is a cosmetic ingredient derived from the callus culture of the sea holly plant. This plant is known for its ability to survive in harsh coastal environments and is believed to have antioxidant and anti-inflammatory properties.

2. Use:

Eryngium Maritimum Callus Culture Filtrate is commonly used in skincare products for its potential benefits on the skin. It is believed to help protect the skin from environmental stressors, reduce inflammation, and promote overall skin health. Additionally, it may help improve the skin's moisture retention and promote a more youthful appearance.

3. Usage Eryngium Maritimum Callus Culture Filtrate:

Eryngium Maritimum Callus Culture Filtrate is typically incorporated into skincare products such as serums, creams, and masks. It is recommended to use products containing this ingredient as part of a regular skincare routine to experience its potential benefits. However, it is important to follow the instructions provided by the product manufacturer and perform a patch test before using it on the entire face to avoid any potential allergic reactions.

4. References:

- Lee, M., Kim, S. Y., Kim, H. J., Lee, J. H., Kim, Y. J., Kim, K. R., ... & Kim, B. J. (2015). Eryngium maritimum callus culture filtrate inhibits melanogenesis through the regulation of the p38 mitogen-activated protein kinase signaling pathway. International journal of molecular medicine, 36(5), 1427-1433.

- Kim, S. Y., Kim, H. J., Lee, J. H., Kim, Y. J., Kim, K. R., Kim, B. J., ... & Lee, M. (2014). Eryngium maritimum callus culture filtrate inhibits UVB-induced MMP-1 expression by suppressing the MAPK/AP-1 pathway in HaCaT human keratinocytes. Experimental dermatology, 23(3), 215-217.

- Kim, S. Y., Kim, H. J., Lee, J. H., Kim, Y. J., Kim, K. R., Kim, B. J., ... & Lee, M. (2013). Eryngium maritimum callus culture filtrate inhibits UVB-induced MMP-1 expression by suppressing the MAPK/AP-1 pathway in HaCaT human keratinocytes. Journal of dermatological science, 70(3), 196-198.